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primary antibodies against tnfsf14, hvem  (Santa Cruz Biotechnology)


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    Santa Cruz Biotechnology primary antibodies against tnfsf14, hvem
    Tnfsf14 deficiency ameliorates UUO-induced renal fibrosis. Kidney tissues from Tnfsf14 +/+ and Tnfsf14 −/− mice were collected at 7 days after UUO surgery. Sham group was used as the control of UUO. ( A ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( B <t>)</t> <t>α-SMA</t> expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( C ) Western blot analyses of renal fibronectin <t>and</t> <t>α-SMA</t> expression in kidney tissues. Representative western blot (Left) and quantitative data (Right) are presented. ( D ) The mRNA levels of pro-fibrotic mediators Cola1 , Vim , and TGF-β1 were measured by qRT-PCR. The data were representative of the results of three independent experiments. All values are represented as mean ± SEM. n = 5 per group. ** P < 0.01 and * ** P < 0.001.
    Primary Antibodies Against Tnfsf14, Hvem, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/primary+antibodies+against+hvem/primary+antibodies+against+tnfsf14++hvem/pmc07803499-130-22-42
    Average 90 stars, based on 1 article reviews
    primary antibodies against tnfsf14, hvem - by Bioz Stars, 2026-09
    90/100 stars

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    1) Product Images from "Tumor necrosis factor superfamily 14 is critical for the development of renal fibrosis"

    Article Title: Tumor necrosis factor superfamily 14 is critical for the development of renal fibrosis

    Journal: Aging (Albany NY)

    doi: 10.18632/aging.104151

    Tnfsf14 deficiency ameliorates UUO-induced renal fibrosis. Kidney tissues from Tnfsf14 +/+ and Tnfsf14 −/− mice were collected at 7 days after UUO surgery. Sham group was used as the control of UUO. ( A ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( B ) α-SMA expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( C ) Western blot analyses of renal fibronectin and α-SMA expression in kidney tissues. Representative western blot (Left) and quantitative data (Right) are presented. ( D ) The mRNA levels of pro-fibrotic mediators Cola1 , Vim , and TGF-β1 were measured by qRT-PCR. The data were representative of the results of three independent experiments. All values are represented as mean ± SEM. n = 5 per group. ** P < 0.01 and * ** P < 0.001.
    Figure Legend Snippet: Tnfsf14 deficiency ameliorates UUO-induced renal fibrosis. Kidney tissues from Tnfsf14 +/+ and Tnfsf14 −/− mice were collected at 7 days after UUO surgery. Sham group was used as the control of UUO. ( A ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( B ) α-SMA expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( C ) Western blot analyses of renal fibronectin and α-SMA expression in kidney tissues. Representative western blot (Left) and quantitative data (Right) are presented. ( D ) The mRNA levels of pro-fibrotic mediators Cola1 , Vim , and TGF-β1 were measured by qRT-PCR. The data were representative of the results of three independent experiments. All values are represented as mean ± SEM. n = 5 per group. ** P < 0.01 and * ** P < 0.001.

    Techniques Used: Control, Staining, Expressing, Immunohistochemistry, Western Blot, Quantitative RT-PCR

    Sphk1 is critical for UUO-induced kidney fibrosis in mice. ( A ) Sphk1 expression in kidney tissues of Tnfsf14 +/+ mice after UUO surgery for 7 days was measured by immunohistochemistry (upper lane, original magnification ×200; lower lane, original magnification ×400). ( B ) Linear regression showed a close correlation between Sphk1 mRNA expression and Cola1 and Acta2 mRNA expression in kidney tissues of Tnfsf14 +/+ mice after UUO surgery for 7 days. Spearman’s correlation coefficient and P value are shown (n = 10). ( C – E ) After UUO surgery, PF543 (1 mg/kg/day) was injected intraperitoneally for consecutive 7 days, and then kidney tissues were collected from each group. ( C ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( D ) α-SMA expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( E ) Western blot analyses of renal fibronectin and α-SMA protein in kidney tissues. Representative western blot (Left and quantitative data (Right) are presented. Sham group was used as the control of UUO. The data were representative of the results of three independent experiments. All values are represented as means ± SEM. n = 5 per group. *** P < 0.001.
    Figure Legend Snippet: Sphk1 is critical for UUO-induced kidney fibrosis in mice. ( A ) Sphk1 expression in kidney tissues of Tnfsf14 +/+ mice after UUO surgery for 7 days was measured by immunohistochemistry (upper lane, original magnification ×200; lower lane, original magnification ×400). ( B ) Linear regression showed a close correlation between Sphk1 mRNA expression and Cola1 and Acta2 mRNA expression in kidney tissues of Tnfsf14 +/+ mice after UUO surgery for 7 days. Spearman’s correlation coefficient and P value are shown (n = 10). ( C – E ) After UUO surgery, PF543 (1 mg/kg/day) was injected intraperitoneally for consecutive 7 days, and then kidney tissues were collected from each group. ( C ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( D ) α-SMA expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( E ) Western blot analyses of renal fibronectin and α-SMA protein in kidney tissues. Representative western blot (Left and quantitative data (Right) are presented. Sham group was used as the control of UUO. The data were representative of the results of three independent experiments. All values are represented as means ± SEM. n = 5 per group. *** P < 0.001.

    Techniques Used: Expressing, Immunohistochemistry, Injection, Staining, Western Blot, Control

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    Article Snippet: .. Briefly, sections (4-μm) were blocked with 5% BSA for 1 h at room temperature and incubated at 4°C overnight with primary antibodies against HVEM (1:100, Santa Cruz Biotechnology), followed by incubation with horseradish peroxidase-conjugated secondary antibody (1:800; Beyotime, Shanghai, China). .. The results were analyzed using DAB assay kits (ZSGB-BIO, Beijing, China) by two pathologists, who were blinded to the clinicopathological features of the samples.

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    primary antibodies against tnfsf14, hvem  (Santa Cruz Biotechnology)
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    Tnfsf14 deficiency ameliorates UUO-induced renal fibrosis. Kidney tissues from Tnfsf14 +/+ and Tnfsf14 −/− mice were collected at 7 days after UUO surgery. Sham group was used as the control of UUO. ( A ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( B <t>)</t> <t>α-SMA</t> expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( C ) Western blot analyses of renal fibronectin <t>and</t> <t>α-SMA</t> expression in kidney tissues. Representative western blot (Left) and quantitative data (Right) are presented. ( D ) The mRNA levels of pro-fibrotic mediators Cola1 , Vim , and TGF-β1 were measured by qRT-PCR. The data were representative of the results of three independent experiments. All values are represented as mean ± SEM. n = 5 per group. ** P < 0.01 and * ** P < 0.001.
    Primary Antibodies Against Tnfsf14, Hvem, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/primary+antibodies+against+hvem/primary+antibodies+against+tnfsf14++hvem/pmc07803499-130-22-42
    Average 90 stars, based on 1 article reviews
    primary antibodies against tnfsf14, hvem - by Bioz Stars, 2026-09
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    Tnfsf14 deficiency ameliorates UUO-induced renal fibrosis. Kidney tissues from Tnfsf14 +/+ and Tnfsf14 −/− mice were collected at 7 days after UUO surgery. Sham group was used as the control of UUO. ( A ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( B <t>)</t> <t>α-SMA</t> expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( C ) Western blot analyses of renal fibronectin <t>and</t> <t>α-SMA</t> expression in kidney tissues. Representative western blot (Left) and quantitative data (Right) are presented. ( D ) The mRNA levels of pro-fibrotic mediators Cola1 , Vim , and TGF-β1 were measured by qRT-PCR. The data were representative of the results of three independent experiments. All values are represented as mean ± SEM. n = 5 per group. ** P < 0.01 and * ** P < 0.001.
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    Tnfsf14 deficiency ameliorates UUO-induced renal fibrosis. Kidney tissues from Tnfsf14 +/+ and Tnfsf14 −/− mice were collected at 7 days after UUO surgery. Sham group was used as the control of UUO. ( A ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( B ) α-SMA expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( C ) Western blot analyses of renal fibronectin and α-SMA expression in kidney tissues. Representative western blot (Left) and quantitative data (Right) are presented. ( D ) The mRNA levels of pro-fibrotic mediators Cola1 , Vim , and TGF-β1 were measured by qRT-PCR. The data were representative of the results of three independent experiments. All values are represented as mean ± SEM. n = 5 per group. ** P < 0.01 and * ** P < 0.001.

    Journal: Aging (Albany NY)

    Article Title: Tumor necrosis factor superfamily 14 is critical for the development of renal fibrosis

    doi: 10.18632/aging.104151

    Figure Lengend Snippet: Tnfsf14 deficiency ameliorates UUO-induced renal fibrosis. Kidney tissues from Tnfsf14 +/+ and Tnfsf14 −/− mice were collected at 7 days after UUO surgery. Sham group was used as the control of UUO. ( A ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( B ) α-SMA expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( C ) Western blot analyses of renal fibronectin and α-SMA expression in kidney tissues. Representative western blot (Left) and quantitative data (Right) are presented. ( D ) The mRNA levels of pro-fibrotic mediators Cola1 , Vim , and TGF-β1 were measured by qRT-PCR. The data were representative of the results of three independent experiments. All values are represented as mean ± SEM. n = 5 per group. ** P < 0.01 and * ** P < 0.001.

    Article Snippet: Briefly, sections were blocked with 5 % BSA for 1 h at room temperature and incubated at 4° C overnight with primary antibodies against α-SMA, LTβR, Sphk1 (All diluted by 1:150; Abcam, Cambridge, MA, USA), and TNFSF14, HVEM (All diluted by 1:150; Santa Cruz, Dallas, TX, USA).

    Techniques: Control, Staining, Expressing, Immunohistochemistry, Western Blot, Quantitative RT-PCR

    Sphk1 is critical for UUO-induced kidney fibrosis in mice. ( A ) Sphk1 expression in kidney tissues of Tnfsf14 +/+ mice after UUO surgery for 7 days was measured by immunohistochemistry (upper lane, original magnification ×200; lower lane, original magnification ×400). ( B ) Linear regression showed a close correlation between Sphk1 mRNA expression and Cola1 and Acta2 mRNA expression in kidney tissues of Tnfsf14 +/+ mice after UUO surgery for 7 days. Spearman’s correlation coefficient and P value are shown (n = 10). ( C – E ) After UUO surgery, PF543 (1 mg/kg/day) was injected intraperitoneally for consecutive 7 days, and then kidney tissues were collected from each group. ( C ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( D ) α-SMA expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( E ) Western blot analyses of renal fibronectin and α-SMA protein in kidney tissues. Representative western blot (Left and quantitative data (Right) are presented. Sham group was used as the control of UUO. The data were representative of the results of three independent experiments. All values are represented as means ± SEM. n = 5 per group. *** P < 0.001.

    Journal: Aging (Albany NY)

    Article Title: Tumor necrosis factor superfamily 14 is critical for the development of renal fibrosis

    doi: 10.18632/aging.104151

    Figure Lengend Snippet: Sphk1 is critical for UUO-induced kidney fibrosis in mice. ( A ) Sphk1 expression in kidney tissues of Tnfsf14 +/+ mice after UUO surgery for 7 days was measured by immunohistochemistry (upper lane, original magnification ×200; lower lane, original magnification ×400). ( B ) Linear regression showed a close correlation between Sphk1 mRNA expression and Cola1 and Acta2 mRNA expression in kidney tissues of Tnfsf14 +/+ mice after UUO surgery for 7 days. Spearman’s correlation coefficient and P value are shown (n = 10). ( C – E ) After UUO surgery, PF543 (1 mg/kg/day) was injected intraperitoneally for consecutive 7 days, and then kidney tissues were collected from each group. ( C ) Sirius Red and Masson staining of kidney tissues sections. Original magnification ×400. ( D ) α-SMA expression in kidney tissues was measured by immunohistochemistry. Original magnification ×200. ( E ) Western blot analyses of renal fibronectin and α-SMA protein in kidney tissues. Representative western blot (Left and quantitative data (Right) are presented. Sham group was used as the control of UUO. The data were representative of the results of three independent experiments. All values are represented as means ± SEM. n = 5 per group. *** P < 0.001.

    Article Snippet: Briefly, sections were blocked with 5 % BSA for 1 h at room temperature and incubated at 4° C overnight with primary antibodies against α-SMA, LTβR, Sphk1 (All diluted by 1:150; Abcam, Cambridge, MA, USA), and TNFSF14, HVEM (All diluted by 1:150; Santa Cruz, Dallas, TX, USA).

    Techniques: Expressing, Immunohistochemistry, Injection, Staining, Western Blot, Control